Tuesday, August 28, 2012

MOMS & DADS:

Please Educate Yourself On These Toxic Study Conclusions!

Hundreds of published studies conclude that maternal consumption of soy transfers multiple soy toxins to developing fetus. Maternal consumption of soy while breast feeding again transfers soy toxins to nursing child. Soy infant formulas are repeatedly proven as highly risky to your child's health. BPA plastics in combination with soy, a double dose of hormone disruptors, add fuel to the fire of increasing the risk of adverse effects to your child. It is a matter of study facts that each, and all exposures to soy phyto-toxins are proven as an extreme risk to your child's body and brain health.


Maternal Diet Transfers Soy-Estrogenic Endocrine Disruptors (genistein, daidzein and glycitein), to Fetus:

2001, FDA NIEHS, The developing fetus is uniquely sensitive to perturbations with estrogenic chemicals. DES is the classic example. Phytoestrogen use in nutritional and pharmaceutical applications for infants and children is increasing. We investigated the carcinogenic potential of genistein, a naturally occurring plant estrogen in soy. At 18 months (mice study) the incidence of uterine adenocarcinoma was 35% for genistein and 31% for DES. Data suggest that genistein is carcinogenic if exposure occurs during critical periods of differentiation. Use of soy–based infant formulas in the absence of medical necessity and the marketing of soy products that appeal to children should be closely examined. www.ncbi.nlm.nih.gov/pubmed/11389053

2001, FDA NIEHS scientists report; Genistein, the principal soy isoflavone has estrogenic activity and is widely consumed by humans. These studies show that genistein aglycone crosses the rat placenta and can reach fetal brain from maternal serum genistein levels that are relevant to those observed in humans. www.ncbi.nlm.nih.gov/pubmed/11297868

1993, Fetal thyroid hormones play an essential role in fetal brain development. It is possible that maternal soy hormone disruptor consumption influence fetal brain development. There is a “critical period” in which appropriate thyroid hormone levels are absolutely essential for normal brain development. In humans this period is considered to begin late in gestation and extend through 1-3 years of age. Deficiencies of thyroid hormone during this critical period cause serious damage to the structural development and organization of the brain. Edrv.endjournals.org/content/14/1/94/short

2009, Autism is probably attributable to a combination of a common genetic background and a possible prenatal exposure or alteration in fetal environment during pregnancy. www.ncbi.nlm.nih.gov/pubmed/19581261 (Maternal consumption of soy phyto-toxic hormone disruptor exposure is repeatedly proven to contaminate fetal environment).

2007, Our results indicate that dietary genistein appears to negatively affect mouse embryoninc development in vivo by inducing cell apoptosis and inhibiting cell proliferation. www.ncbi.nlm.nih.gov/pubmed/17241527

2006, Toxicological Sciences: Diets high in soy –based products are well known for their estrogenic activity. Genistein the predominant phytoestrogen present in soy is known to interact with estrogen receptors alpha and beta and elicits reproductive effects in developing rodents. It is critical to understand the delivery of free and conjugated genistein across the placenta to the fetus following maternal genistein exposure. Genistein level sin placental tissue was high…. Fetal concentrations of unconjugated genistein following administration of 40mg/kg were above the EC50 for ER beta activation. High placental concentrations of genistein indicate the placenta is a potential target organ for genistein action during gestation. www.ncbi.nlm.nih.gov/pubmed?term=Kinetics of genistein and conjugated metabolites in pregnant Sprague-Dawley rats follwing single and repeated genistein administration.

2002, Early exposure to genistein exerts long-lasting effects on the endocrine and immune systems. Data illustrate that exposure to soy genistein during pregnancy and lactation exerts long-lasting effects on the endocrine and immune systems in adulthood. www.ncbi.nlm.nih.gov/pubmed/12520091

2006, Daidzein and genistein (soy hormone disruptors) are found in higher concentrations than BPA in fetal amniotic fluid. www.ncbi.nlm.nih.gov/pubmed/16112541

2002, Johns Hopkins, Primary goal of this study was to compare effects of perinatal exposure with life-long exposure to genistein, an estrogenic compound in soy on the endocrine and immune system in rat adulthood. These data illustrat4e that exposure to genistein during pregnancy and lactation exerts long-lasting effects on the endocrine and immune systems in adulthood. www.ncbi.nlm.gov/pmc/articles/PMC2039948

2002, Fetal soy exposure; evidence of soy phytoestrogens found in amniotic fluid. www.ncbi.nlm.nih. gov/pubmed/11888703

2005, Phytoestrogens transfer between mother and fetus…..placental transfer…the effects of fetal exposure to phytoestrogens should be studied further. (Japan) www.ncbi.nlm.nih.gov/pubmed/16194669

2009 Taiwan study: Genistein has cytotoxic effects on embryo development. Fetal and infant damage. www.ncbi.nlm.nih.gov/pubmed/19490995

2002, Since dietary phytoestrogens account for a significant proportion of human exposure to potential endocrine modulators, and since the placenta does not represent a barrier to daidzein or related soy estrogenic isoflavones, the consequences of these exposures early in life should be examined and monitored carefully. www.ncbi.nlm.nih.gov/pubmed/11875621

2011, Fetal exposure to phytoestrogens can affect the development and function of the male reproductive system www.ncbi.nlm.nih.gov/pubmed/21624456

2011, NIH web site: Effects of genistein in maternal diet on reproductive development and spatial learning in male rats.. exposure to pregnant females is toxic to multiple organs and reproductive behavior in male offspring…. Also alters learning and memory. Sex based differences in behavior….. sensitive to endo disruptionwww.ncbi.nlm.nih.gov/pmc/articles/PMC2834867/

2010, Soy phyto-estrogens and male reproductive function. Cautionsoy formula should be avoided www.ncbi.nlm.nih.gov/pubmed/19919579

2007, Exposure to soy phytoestrogens in perinatal period affects androgen secretion by testicular leydig cells in adult rat. Phytoestrogens have ability to regulate Leydig cells. www.ncbi.nlm.nih.gov/pubmed/17569756

2000, Maternal vegetarian diet associated with hypospadias......soy phytoestrogens have a deleterious effect on the developing male reproductive system. www.ncbi.nlm.nih.gov/pubmed/10619956

2011, Endocrine disruptors and soy genistein damage to male urethral development. www.ncbi.nlm.nih.gov/pubmed/21421236

2001, Neonatal exposure to genistein reduces expression of estrogen receptor alpha and androge4n receptor in testes of adult mice and affects male reproductive organs at molecular levels in adulthood. www.ncbi.nlm.nih.gov/pubmed/11873863

2007 Concern has been raised of potential adverse effects due to the estrogenic and other activities of isoflavones. To assess the teratogenic and fetal toxic potential of genistein several rat studies were conducted. Treatment related anomalies were observed at concentrations of > or =10microg and 100 microg/mL, all embryos were malformed. Adverse effects in the pups were observed. On basis of definitive Prenatal development study the NOAEL for maternal toxicity and adverse effects on embryonic development was considered to be 100mg/kg/day when administered orally by dietary admix. www.ncbi.nlm.nih.gov/pubmed/17433519

2004, In utero (fetal) exposure to genistein: There is pronounced ductal hyperplasia, lactational changes, and fibrosis were observed in mammary glands from genistein group. Postnatal exposure to pharmacological levels of genistein induces profound morphological changes in the mammary glands of adult female rats. There is also potential of genistein to modulate toxicological effects of other toxicant mixtures. Effects of in utero exposure to environmental toxicants and potential interaction with postnatal genistein there is enlargement of the thoracic mammary glands observed in female rat offspring at 200 days of age. www.ncbi.nlm.nih.gov/pubmed/14514955

2011, Developmental exposure to estrogenic compounds can disrupt sexual differentiation in adult reproductive function in many animals including humans. Phytoestrogens in the diet comprise a significant source of estrogenic exposure to humans, particularly infants who are fed soy-based infant formula. Studies clearly demonstrate that environmentally relevant doses of genistein have significant negative impacts on ovarian differentiation, estrous cyclicity, and fertility in the rodent model. Additional studies of reproductive function in human populations exposed to phytoestrogens during development are warranted. www.ncbi.nlm.nih.gov/pubmed/20955782

2006, Dietary component, such as fat or phytochemicals in plant foods, can have an opposite effect on breast cancer risk if exposed in utero through a pregnant mother or at puberty. Dietary exposure during pregnancy often has similar effects on breast cancer risk among mothers and their female offspring. There is extensive programming of the mammary gland during fetal life and subsequent reprogramming at puberty and pregnancy. Thus, dietary exposure during pregnancy and puberty may play an important role in determining later risk by inducing epigenetic changed that modify vulnerability to breast cancer. www.ncbi.nlm.nih.gov/pubmed/17261753

2011, Isoflavones are non-nutritive components of soy responsible for estrogenic responses. There is evidence of potential adverse effects e.g., stimulation of estrogen-dependent mammary tumors and aberrant peri-natal development. Studies of the major soy isoflavone genistein were conducted in pregnant and lactating rats to quantify placental and lactational transfer to plasma and brain to better understand biological effects observed in multi-generational studies. The information derived from these studies also makes it possible to predict internal exposures of children to genistein from soy infant formula. www.ncbi.nlm.nih.gov/pubmed/21034763

2002, Frequency of feedings with soy-based milk formulas in early life was significantly higher in children with autoimmune thyroid disease. Inappropriate thyroid hormone levels can also have a devastating effect on the developing human brain especially during the first 12 weeks of pregnancy when the fetus depends on the mother’s thyroid hormones for brain development. www.rense.com/general3/soy.htm

2010, Serious malformation and a higher abnormal frequency of the central nervous system were induced by the combination of BPA and Genistein. Our findings suggest that genistein is embryotoxic and teratogenic to humans. BPA alone may not be a potential teratogen, but these two estrogenic chemicals have a synergistic effect on emybryonic development when present together during the critical period of major organ formation. Pregnant women should not take soy supplements. www.ncbi.nlm.nih.gov/pubmed/20505512

2004, In utero and lactational exposure to estrogenic agents has been shown to influence morphological and functional development of reproductive tissues. Experiments demonstrate that developmental exposure to dietary isoflavones, at levels comparable to the ranges of human exposure, modify expression of the estrogen-regulated progesterone receptor (PR) in the uterus of sexually mature rats weeks after exposure ended. Since the PR is essential for regulating key female reproductive processes, such as uterine proliferation, implantation, and maintenance of pregnancy, its increased expression suggests that soy phytoestrogen exposure during reproductive development may have long-term reproductive consequences. www.ncbi.nlm.nih.gov/pubmed/14709783


Breast Feeding (Lactational) Transfer Of Soy Toxins to Baby:

2005, Although circulating isoflavone levels are highest among infants consuming soy formula, the fraction of bioavailable isoflavones may be higher in breast-fed infants with mothers who regularly consume soy. Both phytoestrogens and synthetic endocrine disruptors have been found to impair similar reproductive and neuroendocrine endpoints, including sexual differentiation and maturation, fertility, malformation of the genital tract, and sexual behavior suggesting that they have similar mechanisms of action. Indeed both can act as either estrogen agonists or antagonists, depending upon dose, timing of exposure, tissue type, gender, and species. (Exert from, Endocrine Disruptors, Effects on male and Female Reproductive Systems, Second Edition, by Rajesh K. Naz).

2007, Institute of Food Safety, China. Effects of lactational exposure to soy isoflavones on ovary development in neonate rats. Lactational exposure to soy isoflavones induced adverse effects on ovary development in neonate rats, which mechanisms may, at least, particularly involve the modification of mRNA transcription for estrogen receptor and progesterone receptor. www.ncbi.nlm.nih.gov/pubmed/18095567

2008, Lactational exposure to soy could result in estrogen-like actions on female reproductive system www.ncbi.nlm.nih.gov/pubmed/18047477

2008, Genistein passed from the lactating mother to the suckling offspring at levels sufficient to activate gene expression in the reproductive and nonreproductive tissues of mouse pups. In the liver, Estrogen Receptor-alpha and ER-beta messengers RNA and two target genes, CYP17 and the progesterone receptor were modulated by soy genistein. ER-alpha protein level followed an opposite regulation by genistein and estradiol. www.ncbi.nlm.nih.gov/pubmed/18281260

2010, Teratogenic effects and fetal toxicity of environmental estrogenic endocrine disruptors have become a great concern in recent years. Combination bisphenal A (BPA) and Genistein on rat embryos are investigated. Both BPA and Genistein produced concentration-dependent inhibition of embryonic development. Analyses reveal a significant synergistic interaction between BPA and Genistein for most end points, as indicated by enhanced developmental toxicity of BPA after coexposure with Genistein. Serious malformations and higher abnormal frequency of the central nervous system were induced by the combination BPA and Genistein. Findings suggest that Genistein may be embryotoxic and teratogenic to humans. These two estrogenic chemicals have a synergistic effect on embryonic development. Pregnant women should not take soy supplements. www.ncbi.nlm.nih.gov/pubmed/20299547


Soy Infant Formulas Cause Severe Adverse Effects:

1989, This observation confirms previous results obtained with polio diphtheria tetanus and pertussis vaccines indicating that soy-protein formulas may interfere with immunization processes. www.ncbi.nlm.nih.gov/pubmed/2556883

1998, Exposure to estrogenic compound may pose a developmental hazard to infants. Phytoestrogen content of soy infant formulas were 87+/-3 and 49+/-2 microg/g. The phytoestrogen content of cereals varied with brain, with genistein ranging from 3 to 287 microg/g and daidzein from 2-276microg/g. Supplementing the diet of 4-month old infants with a single daily serving of cereal can increase their isoflavone intake by over 25% depending on the brain chosen. This rate of soy isoflavone intake is much greater than that shown in adult humans to alter reproductive hormones. Available evidence suggests that infants can digest and absorb dietary phytoestrogens in active forms and neonates are generally more susceptible than adults to perturbations of the sex steroid milieu, it would desirable to study the effects of soy isoflavone on steroid-dependent developmental processes in human babies. www.ncbi.nlm.nih.gov/pubmed/9402332

2001, FDA NIEHS, The developing fetus is uniquely sensitive to perturbations with estrogenic chemicals. DES is the classic example. Phytoestrogen use in nutritional and pharmaceutical applications for infants and children is increasing. We investigated the carcinogenic potential of genistein, a naturally occurring plant estrogen in soy. At 18 months (mice study) the incidence of uterine adenocarcinoma was 35% for genistein and 31% for DES. Data suggest that genistein is carcinogenic if exposure occurs during critical periods of differentiation. Use of soy –bead infant formulas in the absence of medical necessity and the marketing of soy products that appeal to children should be closely examined. www.ncbi.nlm.nih.gov/pubmed/11389053

2007, FDA, NIEHS report; Soy genistein causes deleterious effects on the developing female reproductive system in adulthood. Altered ovarian differentiation, disrupted ovarian function and estrous cyclicity caused by neonatal exposure, reduced fertility altered mammary gland and behavioral endpoints. Further trangenerational effects were observed in neonatal treatment with genistein at environmentally relevant doses caused adverse consequences on female mice development which is manifested in adulthood. Feeding genistein found in soy formula are similar to those obtained from injecting genistein in mice. www.ncbi.nlm.nih.gov/pubmed/17604387

2011, Compared with girls fed non-soy-based infant formula, soy-fed girls were at 25% higher risk of early menarche throughout the course of follow-up. ….observable manifestation of mild endocrine-disrupting effects of soy isoflavone exposure. www.ncbi.nlm.nih.gov/pubmed/22324503

2010, Soy-based infant formula contain high levels of the estrogenic isoflavone genistein leading to “concern” that neonatal genistein exposure could cause acute and/or long-term adverse effects on reproductive and other organs. Neonatal genistein treatment caused increased relative uterine weight, down-regulation of progesterone receptor in uterine epithelia, genistein was also seen in the neonatal ovary, and thymus, which had an increase in the incidence of multioocyte follicles (MOF) and decrease in thymic weight. Increased MOF’s persisted into adulthood in neonatally treated genistein females. www.ncbi.nlm.nih.gov/pubmed/20357267

2009, French Warn: soy products, in any amount should not be eaten by children under 3 years of age, or women who have breast cancer or at risk of the disease. Israeli Health Ministry: Issued public warning on soy, suggesting soy consumption be limited in young children and avoided if possible in infants. Germany reports: there is a lack of evidence to confirm the safety of soy isoflavone supplements. www.tyroid.about.com/cs/soyinfo/a/soy_4.htm

2009, FDA NIEHS, Developmental exposure to environmental estrogens is associated with adverse consequences later in life. Exposure to genistein, the gylcosylated for of the phytoestrogen genistein found in soy products is of “concern” because U.S. infants are fed soy formula. Exposure to genistein during neonatal life adversely affects the female reproductive system. www.ncbi.nlm.nih.gov/pubmed/20049207

2008, One of eight infants during the first 6 months of life given soy formula may be at risk for brain and behavioral disorders not evident until adolescence, a charge denied by the soy industry. www.soyonlineservice.co.nz/articles/goodman.htm

2010, Soy As An Endocrine Disruptor: Cause For Caution: Endocrine disrupting compounds (EDCs_ alter the function of the endocrine system and consequently cause adverse health effects. Phytoestrogens, natural plant compounds abundantly found in soy and soy products, behave as weak estrogen mimics or as anti-estrogen. They are considered to be EDCs. Supporting evidence that consumption of phytoestrogens is beneficial is indirect and inconsistent. Lifetime exposure to estrogenic substances, especially during critical periods of development, has been associated with formation of malignancies and several anomalies of the reproductive systems. Phytoestrogen consumption in infants, through soy-based formulas is of particular concern. Possible adverse effects should not be taken lightly. www.ncbi.nlm.nih.gov/pubmed/21175082

2001 Studies show that soybean-based formulas contain large quantities of phytoestrogens, particularly isoflavones. Because of experimental data suggesting a possible deleterious effect of phytoestrogens on the neuro-endocrine maturation, the reduction of soy content in formulas must be considered. www.ncbi.nlm.nih.gov/pubmed/11760676

2010 Uterine fibroids are the most common pelvic tumors in U.S. women as well as the most common cause for hysterectomy. The results showed an association between early fibroid diagnosis and having been fed soy formula during infancy. www.ncbi.nlm.nih.gov.pmc/articles/PMC2854791

2009 Bulletin of Academy of National Medicine. Infant Nutrition Nutritional quality during the first weeks of life can influence health during both infancy and adulthood. Exclusive long-term breast feeding is strongly recommended….Soy-based formulas are not recommended for healthy infants. www.ncbi.nlm.nih.gov/pubmed/19718896

2010 Soy isoflavones, genistein and daidzein are widely consumed with evidence for potential adverse effects. Study results show that soy protein isolate is an efficient isoflavone delivery vehicle capable of providing significant proportions of the total dose into the circulation in the active aglycone form for distribution to hormone receptor-bearing tissues and subsequent pharmacological effects that determine possible health risks. www.ncbi.nlm.nih.gov/pubmed/20225898

2011, Compared with girls fed non-soy-based infant formula or milk formula, early soy-fed girls were at 25% higher risk of menarche. Results also suggest that girls fed soy products in early infancy may have an increase risk of menarche specifically in early adolescence and may be the manifestation of mild endocrine disrupting effects o f soy isoflavone exposure. This soy formula association with menarche warrants more in-depth evaluation. www.ncbi.nlm.nih.gov/pubmed/22324503

2003, Soy infant formula and phytoestrogens: Soy infant formula contains high levels of the isoflavones, genistein and daidzein which are referred to as phytoestrogens, and are structurally similar to estrogen. Infants consuming soy have high levels of circulating isoflavones, grater than the levels of isoflavones which have been shown to produce physiological effects in adult women consuming a high soy diet. Most would argue for a “precautionary approach to be taken where there are potential developmental effects from the consumption of pharmacologically active compounds.www.ncbi.nlm.nih.gov/pubmed/12919490

2010- Acute and Chronic Effects of soy…raise concerns that high genistein are estrogenic and impact development of human infants fed soy formula. www.ncbi.nlm.nih.gov/pubmed/20357267

2001, NIEHS report: Dietary (soy) genistein produced effects in multiple estrogen-sensitive tissues in males and females consistent with estrogenic activity….within exposure ranges in humans. www.ncbi.nlm.nih.gov/pubmed/11738518

2002, Early soy exposure causes long lasting adverse effects later in life: www.ncbi.nlm.gov/pmc/articles/PMC2039948/

2002, Frequency of feedings with soy-based milk formulas in early life was significantly higher in children with autoimmune thyroid disease. Inappropriate thyroid hormone levels can also have a devastating effect on the developing human brain especially during the first 12 weeks of pregnancy when the fetus depends on the mother’s thyroid hormones for brain development. www.rense.com/general3/soy.htm

2006, Estrogen regulates thymic development and immune function. Genistein administration to mice that produced serum genistein concentrations similar to those reported in human infants consuming soy formula and had demonstrable effects. Genistein had similar effects on many estradiol target genes, affecting genes involved in transcription, apoptosis, cell cycle and thymic development and function. www.ncbi.nlm.nih.gov/pubmed/16484547

2010 NIEHS study: Acute and Chronic effects of soy genistein in neonatal mice… Immediate and long-term effects raise concerns that genistein are estrogenic and impact development of human infants fed soy formula. Increase uterine weight down-regulation of progesterone receptor in uterus, neonatal ovary and thymus decrease thymic weight…genistein are estrogenic and impact human infants fed soy formula. www.ncbi.nlm.nih.gov/pubmed/20357267

2010, Soy-based infant formula contain high levels of the estrogenic isoflavone genistein leading to “concern” that neonatal genistein exposure could cause acute and/or long-term adverse effects on reproductive and other organs. Neonatal genistein treatment caused increased relative uterine weight, down-regulation of progesterone receptor in uterine epithelia, genistein was also seen in the neonatal ovary, and thymus, which had an increase in the incidence of multioocyte follicles (MOF) and decrease in thymic weight. Increased MOF’s persisted into adulthood in neonatally treated genistein females. www.ncbi.nlm.nih.gov/pubmed/20357267

2005, In summary, neonatal treatment with genistein caused abnormal estrous cycles, altered ovarian function, early reproductive senescence, and subfertility/infertility at environmentally relevant doses. www.bioreprod.org/cgi/content/abstract/73/4/798

2004, Trypsin inhibitors interfere with protein digestion, cause pancreatic disorders and act as a growth inhibitor. All of this is found in soy infant formula. Hemaglutinin, a clot-causing agent that is dangerous for those with heart disease. The phytic acid content in soybeans is highest of all grains or legumes. Phytic acid can cause mineral deficiencies in iron, copper, zinc, calcium, and magnesium. Zinc is essential for growth of the nervous system and is called the intelligence mineral. Soy is known goitrogen that may cause hypothyroidism, by causing damage to enzymes that produce thyroid hormones. Iodine is blocked with is essential for proper thyroid function. Aluminum content in soy is about 10 times higher than milk, and can directly damage the infant’s brain, as the blood-brain barrier has not been formed. In 1913 the USDA classified soy not as a food but as an industrial product. Why has a product that has only been given GRAS standards for industrial use become a major component of 60% of our supermarket foods? Have we made corporate profits and conveniences more important than health? www.htnetwork/org/articles/soycontroversy.html

2005, Total isoflavone content in soy infant formula varies widely, but in general is quite high with as much as 122ug genistein and 77ug daidzein per gram of formula. This translates to a daily intake of approximately 6-9 mg/kg body weight per day, which is 4-7 times higher than the amounts regularly consumed by adults meeting the FDA guidelines for soy consumption. Infants fed soy formula have circulating phytoestrogen concentrations of approximately 13,000 to 22,000times higher than endogenous estrogens, or levels high enough to produce many of the physiological effects discussed in this chapter. Although circulating isoflavone levels are highest among infants consuming soy formula, the fraction of bioavailable isoflavones may be higher in breast-fed infants with mothers who regularly consume soy. Both phytoestrogens and synthetic endocrine disruptors have been found to impair similar reproductive and neuroendocrine endpoints, including sexual differentiation and maturation, fertility, malformation of the genital tract, and sexual behavior suggesting that they have similar mechanisms of action. Indeed both can act as either estrogen agonists or antagonists, depending upon dose, timing of exposure, tissue type, gender, and species. (Exert from, Endocrine Disruptors, Effects on male and Female Reproductive Systems, Second Edition, by Rajesh K. Naz).

2010, Soy isoflavone, genistein and daidzein, are widely consumed in soy-based foods, evidence for potential adverse effects has been obtained from experimental animal studies. These results show that soy protein isolate (used in soy infant formula) is an efficient isoflavone delivery vehicle capable of providing significant proportions of the total dose into the circulation in the active aglycone form for distribution to receptor-bearing tissues and subsequent pharmacological effects that determine possible health risks. www.ncbi.nlm.nih.gov/pubmed/20225898

New Zealand speaks out against soy formula as serious risks are caused by soy endocrine disruptors. www.kidalog.net/soyformula.html

ALARMING NOTE: Read the ingredients, the FDA is allowing soy industry to add soy phyto-toxins to infant milk formulas, and multiple infant foods while well-aware of soy's highly toxic cause of adverse health effects!


BPA and Soy Combination = Highly Toxic Cocktail:

2011, Bisphenol A (BPA) and Genistein, the predominant component of soy products are both known environmental estrogens. We investigated the developmental toxicity of BPA and Genistein and their combined effects. Genistein as a teratogen was solid… The combined effect of BPA and Genistein was generally additive action… www.ncbi.nlm.nih.gov/pubmed/21034807

1997, In addition to exposure to man-made chemicals (pesticides, polychlorinated biphenyls, phenolic compounds, phthalate esters, and organochlorine), the consumption of plant-derived estrogens in foodstuffs poses a potential risk to human health as phytoestrogens are more potent estrogens and their intake by some infants is likely to be considerable. www.ncbi.nlm.nih.gov/pubmed/9414467

2010, Serious malformation and a higher abnormal frequency of the central nervous system were induced by the combination of BPA and Genistein. Our findings suggest that genistein is embryotoxic and teratogenic to humans. BPA alone may not be a potential teratogen, but these two estrogenic chemicals have a synergistic effect on emybryonic development when present together during the critical period of major organ formation. Pregnant women should not take soy supplements. www.ncbi.nlm.nih.gov/pubmed/20505512

2007, NIEHS study: We further demonstrated that neonatal exposure to the endocrine active compounds (EAC's) genistein and Bisphenol-A (BPA) can affect sexually dimorphic brain morphology and neuronal phenotypes in adulthood with regional and cellular specificity. Developmental EAC exposure has been show to affect a variety of sexually dimorphic behaviors including reproductive behavior.Maladaptive behavior could translate to decreased fitness of entire populations. www.ncbi.nlm.nih.gtov/pubmed/17822772


In truth, soy is loaded with multiple toxins capable of severely damaging physiological, reproductive, and neurological health for all people, and especially developing fetus, infants, and children. For additional soy phyto-toxic study evidence proving the cause of disease and disorders look at:

http://causingcancers.blogspot.com

http://genderchaos.blogspot.com

http://reproductivedefects.blogspot.com

http://neurodefects.blogspot.com

http://soyhypothyroidism.blogspot.com

Overwhelming study evidence repeatedly proves the FDA is protecting a highly powerful U.S. soy phyto-toxic multi-billion dollar industry, over and above their acknowledgement of soy-causation of human pain and suffering from severe and fatal disease. Is this NOT a crime?

What can be done to STOP the FDA from knowingly and willingly concealing soy-poisoning from a trusting American public? An FDA investigation and accountability is long past due!

Gail Elbek

Investigative researcher

gaelbek@yahoo.com

@SoySorry